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Airway obstruction may be total or partial. Total obstruction is characterized by the lack of any air movement or breath sounds. Confronted with ineffective breathing efforts, it is important that inexperienced persons not interpret any retractive movements of the rib cage and the diaphragmatic tugging motions as respiration. Actual air movement must be perceived by feeling with the hand or placing the ear over the mouth. Recognition of obstruction depends on close observation and a high index of suspicion.
The patient with partial obstruction exhibits diminished tidal exchange that is associated with retraction of the upper chest and accompanied by a snoring sound if the obstruction is nasopharyngeal or by inspiratory stridor if obstruction is near the area of the larynx. If inspiratory efforts are severe, the upper airway may undergo a dynamic inspiratory compression because of the marked pressure gradient in the upper airway.
Treatment of upper airway obstruction depends for the most part on whether it is caused by soft tissue obstruction, tumor, foreign body, or laryngospasm. Most often, upper airway obstruction is caused by a reduction of the space between the pharyngeal wall and the base of the tongue by relaxation of the tongue and jaw. The same obstruction may occur with foreign bodies or even with dentures. In the absence of a foreign body, airflow may be restored by preventing the mandible from falling back. Forward motion is applied by placing the forefinger and second finger behind the angle of the mandible.
One of the primary skills required of anesthesiologists is the ability to correct upper airway obstruction in the unconscious or anesthetized patient. This obstruction is commonly attributed to occlusion of the pharynx by the tongue, which falls back. Correction of such a problem would be expected by insertion of devices called oropharyngeal or nasopharyngeal airways (insertion is described later in the chapter). The nasopharyngeal airway is a soft rubber tube that is less traumatic and better tolerated in lighter stages of anesthesia or unconsciousness. The airway itself extends sufficiently into the pharynx to pass behind the base of the tongue. In situations in which the nares do not permit passage, bleeding occurs, or obstruction is not perceived, an oropharyngeal airway may be used. The latter is designed for insertion along the tongue until teeth or gums prevent further passage. Placement of these devices that provide an artificial passage behind the tongue does not in many cases provide unobstructed airflow. This suggests that the tongue is not the principal cause of upper airway obstruction in the anesthetized patient.
This idea is further borne out by the common experience of the need to use head tilt and maintain head extension, even in patients with an oral airway in place. It is difficult to understand how this means of establishing airway patency produces any forward pull on the tongue. This maneuver elevates the hyoid and epiglottis in anesthetized patients. Further support of the mandible may be provided by forward traction or pressing forward at the mandibular angles. When these simple maneuvers and the use of artificial airways do not provide adequate relief of upper airway obstruction, the insertion of an endotracheal tube to bypass the upper airway must be contemplated.
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